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There are several mechanisms to halt cancer growth, including induction of cell cycle arrest, inhibition of cell survival and growth signaling pathways such as phosphoinositide 3-kinase (PI3-K)/Akt, c-Jun N-terminal kinases (JNK), and p38 MAPK pathways—which are often aberrant in cancer (Wagner and Nebreda 2009, Samatar and Poulikakos 2014, Noorolyai, Shajari et al
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