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[3] Additional in-vitro studies suggest that GHK-Cu may down-regulate transforming growth factor-β1, a cytokine implicated in premature follicle regression, thereby potentially prolonging the anagen (growth) phase
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Duodenocutaneous fistula in rats as a model for “wound healing-therapy” in ulcer healing: the effect of pentadecapeptide BPC 157, L-nitro-arginine methyl ester and L-arginine
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