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Similarly, mice with constitutive activation of Akt show reduced apoptosis of salivary acinar cells due to inhibition of p53-mediated apoptosis, which was shown to be dependent on Akt-induced phosphorylation and activation of MDM2, leading to p53 ubiquitination and degradation, reduced mRNA and protein levels of the cell cycle regulator p21 and reduced expression of the p53 homologs, p63 and p73 at 24 h post-IR [6]

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