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Nevertheless, because FSTL3 inhibits multiple TGFβ family ligands 111 , cardiac effects of FSTL3 cannot be attributed to a particular ligand interaction at this time

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33, 34, 35, 36 These data contribute to identifying gene interactions and networks that may be helpful in improving our understanding of GH action (although extrapolating our observations from whole blood to other tissues requires a cautious approach)