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Whilst this route is essential for treating pernicious anaemia and other malabsorption conditions, it can produce serum B12 levels far exceeding physiological requirements

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Beyond its classical redox role, NAD+ serves as a substrate for sirtuin deacylases, PARP DNA-repair enzymes, and CD38 NAD-glycohydrolase — three enzyme families whose activities deplete the NAD+ pool and link cellular NAD+ availability to ageing-related research framings