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Accordingly, a recent report documented that brain apoptosis faced an increase in the C16-ceramide levels potentially by the conversion from mitochondrial sphinganine and sphingomyelin (Mignard et al., 2020)
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The difference in the amounts of ROS produced by neutrophils and macrophages may also explain why Gsr deficiency preferentially compromises the phagocytosis of neutrophils (Fig
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